Convicted Child Abuser Complains Of Harsh Jail Conditions, Haredim Move To Back Abuser

Abuser won't eat prison food because food is not vegan.

This appears to be the leader of the infamous Burqa Cult from Beit Shemesh:

Abusive mother complains of harsh prison conditions
Mother convicted of abusing six of her 12 children says she is starving and her cell is full of mildew
Aviad Glickman * Ynet

A mother from Beit Shemesh convicted of abusing six of her twelve children claimed at a court hearing for the remand of her arrest Wednesday that conditions in the prison where she is being held are difficult for her to bear.

The woman is currently awaiting sentencing by Jerusalem's District Court. During the hearing her attorney, Gil Dachuach, presented a statement by the defendant.

"My mattress has mildew, my blanket has mildew, I'm breathing mildew fumes and it's awful," the statement said. "I can't go on this way. My floor is full of sweat puddles and the room is very damp."

The court also heard that the mother's cell is cold, malodorous, and dirty, her bed filthy and fetid, and her sheets repulsive. Dachuach also claims the cell is plagued by mosquitoes and remnants of food.

The mother also spends most of her time shackled by both wrists, and claims she is starving because she is a vegan and therefore refuses to eat the food offered by the prison.

Testimony to this effect was also given by Rabbi Avraham Froelich, who has taken charge of another mother accused of starving her toddler son.

He said the accused had been allotted the cell adjacent to the mother and that after her release she had spoken about the harsh physical conditions faced by the latter. Froelich said he was told the defendant would need rehabilitation following the conditions of her custody and that he may take her under his charge as well.

Dachuach appealed for his client's release under house arrest as the prison was not capable of handling a woman in such poor mental health. 

 
"She is in a very small room, she doesn't go out for walks, she can't phone or have visitors because the family is prohibited from meeting with her, and she is in an extreme emotional state," he said.

However the prosecution told the court that the mother could not be trusted under more lenient conditions. "We must remember that this is a detainee who has been convicted of a very serious and dangerous crime," attorney Dotan Rosso said. "We must prefer the safety of her children over her own interests."

Comments

[phillip]

Why should I care?

[Proton Soup]

"Why should I care?"

veganism is something you should be concerned about. veganism imposed on children is child abuse. it causes nutritional deficiencies such as low cobalamin, iron, zinc, essential fatty acids, and protein. the lack of cobalamin alone can cause impaired mental condition, or lack of development and birth defects.

if she is a part of some cult of veganism there, it is something you will surely want to deal with, lest you see more of this and worse.

[Dorian]

AH! Burka Rebbetzin Bruria Keren and the idiots of the veil..
Here is a link to see the vegan frummie...
I'll send her a carrot if she is so stressed....

http://www.theawarenesscenter.org/keren_bruria.html

[Mr. Apikoros]

Let her eat cheeseburgers.

[Dorian]

ok..but then with some laxative in it...and a broken toilet...without toiletpaper.
Let's see how fast the veils come off..

[ah-pee-chorus]

when is the fundraising dinner?

[HS]

The American Dietetic Association recently released an updated position statement on vegan diets, affirming that veganism is healthy for babies, children, and adults:

"It is the position of the American Dietetic Association that appropriately planned vegetarian diets, including total vegetarian or vegan diets, are healthful, nutritionally adequate, and may provide health benefits in the prevention and treatment of certain diseases. Well-planned vegetarian diets are appropriate for individuals during all stages of the life cycle, including pregnancy, lactation, infancy, childhood, and adolescence, and for athletes..."

The statement goes on to list many of the diseases that vegetarian /vegan lifestyles help prevent.

J Am Diet Assoc. 2009 Jul;109(7):1266-82.

The full text is at http://www.ncbi.nlm.nih.gov/pubmed/19562864?ordinalpos=1&itool=EntrezSystem2.PEntrez.Pubmed.Pubmed_ResultsPanel.Pubmed_DiscoveryPanel.Pubmed_Discovery_RA&linkpos=2&log$=relatedarticles&logdbfrom=pubmed

[Dorian]

"...when is the fundraising dinner?.."

Tomorrow Tursday-night there will be a solidarity dinner for Benizri. Now..if we play it well and share the costs we could send them some 'prepared cheeseburgers' with a kosher lable on it...and one piece of toiletpaper wrapped around it.
Ofcours first sabotaging the toilets..
Don't forget your camera..

[WoolSilkCotton]

The key words are "...appropriately planned vegetarian diets..." which I doubt this pseudoreligious nutjob actually did.

You can indeed lead a very healthy lifestyle as a veggie, but it takes a real effort to educate yourself, buy the right healthy foods, and eat what you should each day for a balanced proper diet.

A diet of lukshen kugel, challah and chocolate cake will qualify you as a vegetarian, but guess what you'll look like after a few weeks.

[Shlomo]

It may theoretically be possible to have a healthy diet for a vegan but it is very difficult even for an adult to do so. Imagine what the children (who usually are very fussy anyway from experience) would have eaten I doubt that she would be rich to afford expensive food.
A question is it my imagination but it seems to me that very frum people tend to be more involved in alternative medicine diets etc

[NotParticularlyFrumButSickOfBS]

She won't eat? Good. Let her die of starvation.

[Yakira]

It is not hard at all to have a very healthy vegetarian diet as an adult or child; furthermore, it costs a lot less money to eat as a vegetarian. I have family members who were born and raised as vegetarians and they live very healthy and energetic lifestyles. Most of the minerals and vitamins that we need on a daily basis has to come from veggies, fruits, and grains. I am a Certified Fitness Trainer and I know many professional athletes who are STRICT VEGETARIANS.

I could care less about this woman's hardships in prison.

[Proton Soup]

as a "certified fitness professional", you should at the very least be aware that vegetarian means something very different from vegan.

i will try to refrain from getting into a pubmed battle with HS, but suffice to say that suffice to say that if you raise your children vegan, they will be less intelligent than their omnivorous peers. better to keep vegan diets where they belong, wealthy people past child-bearing and -rearing age who feel compelled to injure themselves.

[Yakira]

Proton Soup: If you really knew exactly what "vegan" meant, you would not had made that ridicolous comment. There are different types of vegetarians: Total Vegetarians (Eat only plant food. They do not eat any animal foods, including fish, eggs, dairy products, and honey.); VEGANS (omit all animal products from their diets, but they also eliminate them from the rest of their life. Vegans use nothing from animals, such as leather, wool, and silk.); Lacto-vegetarians (include dairy products into their diet of plant food); Lacto-ovo-vegetarians (eat both eggs and dairy products); Pesco-vegetarians (include fish into their diets); and Pollo-vegetarians (eat poultry, such as chicken, turkey, and duck). I HAVE RELATIVES WHO WERE BORNED AND RAISED AS VEGANS, THEY ARE NOT UNHEALTHY IDIOTS as you previously asserted that all Vegans have inferior intelligence. I STRONGLY SUGGEST that you do more research on veganism before passing ignorant judgement.

[Proton Soup]

alright, we can start here. then maybe move on to birth defects and such.

http://www.ajcn.org/cgi/content/full/72/3/762
Am J Clin Nutr. 2000 Sep;72(3):762-9.
Signs of impaired cognitive function in adolescents with marginal cobalamin status.
Louwman MW, van Dusseldorp M, van de Vijver FJ, Thomas CM, Schneede J, Ueland PM, Refsum H, van Staveren WA.

Division of Human Nutrition and Epidemiology, Wageningen Agricultural University, Netherlands.

BACKGROUND: Lack of cobalamin may lead to neurologic disorders, which have been reported in strict vegetarians. OBJECTIVE: The objective of this study was to investigate whether cognitive functioning is affected in adolescents (aged 10-16 y) with marginal cobalamin status as a result of being fed a macrobiotic diet up to an average age of 6 y. DESIGN: Data on dietary intake, psychological test performance, and biochemical variables of cobalamin status were collected from 48 adolescents who consumed macrobiotic (vegan type) diets up to the age of 6 y, subsequently followed by lactovegetarian or omnivorous diets, and from 24 subjects (aged 10-18 y) who were fed omnivorous diets from birth onward. Thirty-one subjects from the previously macrobiotic group were cobalamin deficient according to their plasma methylmalonic acid concentrations. Seventeen previously macrobiotic subjects and all control subjects had normal cobalamin status. RESULTS: The control subjects performed better on most psychological tests than did macrobiotic subjects with low or normal cobalamin status. A significant relation between test score and cobalamin deficiency (P: = 0.01) was observed for a test measuring fluid intelligence (correlation coefficient: -0.28; 95% CI: -0.48, -0.08). This effect became more pronounced (P: = 0.003) within the subgroup of macrobiotic subjects (correlation coefficient: -0.38; 95% CI: -0.62, - 0.14). CONCLUSION: Our data suggest that cobalamin deficiency, in the absence of hematologic signs, may lead to impaired cognitive performance in adolescents.

PMID: 10966896 [PubMed - indexed for MEDLINE]

Med J Aust. 1979 Jul 14;2(1):1-3.Links
Brain damage in infancy and dietary vitamin B12 deficiency.
Wighton MC, Manson JI, Speed I, Robertson E, Chapman E.

A case of the exclusively breast-fed infant of a vegetarian mother is reported. Neurological deterioration commenced between three and six months of age, and progressed to a comatose premoribund state by the age of nine months. Investigations revealed a mild nutritional vitamin B12 deficiency in the mother, and a very severe nutritional B12 deficiency in the infant, with severe megaloblastic anaemia. Treatment of the infant with vitamin B12 resulted in a rapid clinical and haematological improvement, but neurological recovery was incomplete. Evidence is presented that dietary B12 deficiency was the sole cause of the infant's deterioration, and the literature relating to the condition is reviewed. It is recommended that all strict vegetarians (vegans), especially women in the child-bearing age group, take vitamin B12 supplements.

PMID: 502936 [PubMed - indexed for MEDLINE]

Z Geburtshilfe Neonatol. 2007 Aug;211(4):157-61.Click here to read Links
[Maternal vitamin B12 deficiency: cause for neurological symptoms in infancy]
[Article in German]

Lücke T, Korenke GC, Poggenburg I, Bentele KH, Das AM, Hartmann H.

Klinik für Kinder und Jugendliche, Medizinische Hochschule Hannover.

BACKGROUND: Symptoms of Vitamin B (12) deficiency in infancy include growth retardation, regression of psychomotor development, muscular hypotonia and brain atrophy. Besides an inappropriate vegetarian diet of the infants, a vegan diet or a pernicious anaemia of the mother may lead to an insufficient vitamin B (12) supply of the child. PATIENTS AND METHODS: We report here the neurological symptoms of 4 fully breast-fed infants from mothers on vegan diet or with pernicious anaemia. DISCUSSION AND CONCLUSION: Vitamin B (12) deficiency can easily be diagnosed by detection of methylmalonic acid when measuring the organic acids in urine. Vitamin B (12) deficiency should be avoided or diagnosed as early as possible since a supplementation of mother and child can prevent neurological symptoms of the baby. Furthermore, the neurological symptoms of the infant with manifest vitamin B (12) deficiency are (partially) reversible.

PMID: 17729202 [PubMed - indexed for MEDLINE]

http://www.ajcn.org/cgi/content/full/78/1/3
Vegetarianism and vitamin B-12 (cobalamin) deficiency

can malnutrition make moms act crazy? why yes, yes it can.
N Engl J Med. 1988 Jun 30;318(26):1720-8.Links
Neuropsychiatric disorders caused by cobalamin deficiency in the absence of anemia or macrocytosis.
Lindenbaum J, Healton EB, Savage DG, Brust JC, Garrett TJ, Podell ER, Marcell PD, Stabler SP, Allen RH.

Department of Medicine, Columbia-Presbyterian Medical Center, New York, NY 10032.

Among 141 consecutive patients with neuro-psychiatric abnormalities due to cobalamin deficiency, we found that 40 (28 percent) had no anemia or macrocytosis. The hematocrit was normal in 34, the mean cell volume was normal in 25, and both tests were normal in 19. Characteristic features in such patients included paresthesia, sensory loss, ataxia, dementia, and psychiatric disorders; longstanding neurologic symptoms without anemia; normal white-cell and platelet counts and serum bilirubin and lactate dehydrogenase levels; and markedly elevated serum concentrations of methylmalonic acid and total homocysteine. Serum cobalamin levels were above 150 pmol per liter (200 pg per milliliter) in 2 patients, between 75 and 150 pmol per liter (100 and 200 pg per milliliter) in 16, and below 75 pmol per liter (100 pg per milliliter) in only 22. Except for one patient who died during the first week of treatment, every patient in this group benefited from cobalamin therapy. Responses included improvement in neuropsychiatric abnormalities (39 of 39), improvement (often within the normal range) in one or more hematologic findings (36 of 39), and a decrease of more than 50 percent in levels of serum methylmalonic acid, total homocysteine, or both (31 of 31). We conclude that neuropsychiatric disorders due to cobalamin deficiency occur commonly in the absence of anemia or an elevated mean cell volume and that measurements of serum methylmalonic acid and total homocysteine both before and after treatment are useful in the diagnosis of these patients.

PMID: 3374544 [PubMed - indexed for MEDLINE]

Nutr Rev. 1996 Dec;54(12):382-90.Links
Folate, vitamin B12, and neuropsychiatric disorders.
Bottiglieri T.

Kimberly H. Courtwright and Joseph W. Summers Institute of Metabolic Disease, Baylor University Medical Center, Dallas, Texas, USA.

Folate and vitamin B12 are required both in the methylation of homocysteine to methionine and in the synthesis of S-adenosylmethionine. S-adenosylmethionine is involved in numerous methylation reactions involving proteins, phospholipids, DNA, and neurotransmitter metabolism. Both folate and vitamin B12 deficiency may cause similar neurologic and psychiatric disturbances including depression, dementia, and a demyelinating myelopathy. A current theory proposes that a defect in methylation processes is central to the biochemical basis of the neuropsychiatry of these vitamin deficiencies. Folate deficiency may specifically affect central monoamine metabolism and aggravate depressive disorders. In addition, the neurotoxic effects of homocysteine may also play a role in the neurologic and psychiatric disturbances that are associated with folate and vitamin B12 deficiency.

PMID: 9155210 [PubMed - indexed for MEDLINE]

Electromyogr Clin Neurophysiol. 2003 Jan-Feb;43(1):57-64.Links
Vitamin B12 deficiency neurological syndromes: a clinical, MRI and electrodiagnostic study.
Misra UK, Kalita J, Das A.

Department of Neurology, Sanjay Gandhi PGIMS, Lucknow. ukmisra@sgpgi.ac.in

BACKGROUND: Vegetarianism is an important cause of vitamin B12 deficiency, especially in countries like India. We managed 17 patients with neurological syndrome due to vitamin B12 deficiency in a tertiary care referral teaching hospital which caters to relatively affluent population. AIM: To evaluate neurophysiological and MRI changes in patients presenting with vitamin B12 deficiency neurological syndrome and interpret these is the light of reported autopsy findings. SETTING: Tertiary care referral teaching hospital. METHODS: Patients with vitamin B12 deficiency neurological syndrome diagnosed by low serum vitamin B12 and/or megaloblastic bone marrow were subjected to clinical evaluation and spinal MRI. The neurophysiological tests included nerve conduction studies, tibial somatosensory evoked potential (SEP), motor evoked potential (MEP) and visual evoked potential (VEP) studies. The recovery was defined on the basis of 6 months Barthel Index score into complete, partial or poor. RESULTS: There were 17 patients with vitamin B12 deficiency neurological syndrome, 3 were females and 12 lactovegetarian. The clinical syndrome was that of myelopathy in 8, myeloneuropathy in 5, dementia myelopathy in 3 and neuropathy in 1 patient. All the patients had impaired joint position and vibration sensation in the lower limbs and 4 had in upper limbs as well. Lower limbs were spastic in 13 and upper limbs in 2 patients. Spinal MRI revealed T2 hyperintensity in cervicodorsal region in 6 and cord atrophy in 3 patients. Sural nerve conduction was abnormal in 8 and peroneal conduction in 5 patients. In one patient all sensory nerve conductions were unrecordable but motor conductions were normal. Tibial SEP was abnormal in 12 out of 15 and lower limb MEP in 8 out of 12 patients. P100 latency of VEP was prolonged in 7 out of 13 patients. Right to left asymmetry was present in tibial SEP in 4 and VEP in 2 patients. At 6 months followup 2 patients improved completely, 7 partially and 3 had poor recovery. Clinical recovery correlated with MEP but not with SEP or MRI changes. CONCLUSION: The evoked potential and MRI changes in vitamin B12 deficiency neurological syndrome are consistent with focal demyelination of white matter in spinal cord and optic nerve. Myelopathic presentation is commoner and SEP is more frequently abnormal. The outcome at 6 months correlated with MEP changes.

PMID: 12613142 [PubMed - indexed for MEDLINE]

horrors:
http://jama.ama-assn.org/cgi/content/full/289/8/979

Neurologic Impairment in Children Associated With Maternal Dietary Deficiency of Cobalamin—Georgia, 2001

JAMA. 2003;289:979-980.

MMWR. 2003;52:61-64

2 tables omitted

During 2001, neurologic impairment resulting from cobalamin (vitamin B12) deficiency was diagnosed in two children in Georgia. The children were breastfed by mothers who followed vegetarian diets.* This report summarizes the two cases and provides guidance for health-care providers on identifying and preventing cobalamin deficiency among breastfed infants of vegetarian mothers.

Case 1

During August 2001, a girl aged 15 months was hospitalized for lethargy and failure to thrive. She was born after a full-term pregnancy complicated by prolonged nausea and vomiting. She was breastfed for 8 months, but the extent (exclusivity) of breast milk consumed relative to other food was unknown. Her mother reported following a vegan diet during the preceding 7 years and took nutritional and vitamin supplements. The cobalamin content of the supplements was unknown. When the child was aged approximately 8 months, organic whole-grain cereals and fruit shakes were introduced, but she had a poor appetite and vomited regularly. Her parents became concerned about her growth and development, and she was evaluated by a pediatrician at age 15 months. The pediatrician diagnosed failure to thrive, developmental delay, and severe macrocytic anemia. The child was hospitalized, and cobalamin deficiency was diagnosed (marked elevation [not quantified] of urine methylmalonic acid; serum B12:100 pg/mL [normal range: 210-911 pg/mL]).

The child received supplementary food by mouth and by nasogastric tube. She also received 2 mg of cyanocobalamin and 3 mg of hydroxocobalamin intramuscularly (IM) over 3 days. Three days later, she had partial complex seizures, which stopped without anticonvulsants. A brain MRI indicated global cerebral atrophy. The mother was treated with 1 mg of cobalamin IM.

At age 16 months, the child was seen in a genetics clinic to eliminate possible genetic causes of her neurologic deficiency. At age 28 months, her developmental skills ranged from 9 months for fine motor skills to 18 months for gross motor skills. Her expressive language was at 10 months, and her receptive language was at 12 months. At age 32 months, she had made developmental progress but continued to have developmental delays, especially in speech and language. She was prescribed daily sublingual cobalamin supplements.
...

i strongly suggest you do more than wave your little fitness trainer certificate in front of me before suggesting that I do my research.

idiot